Feline ureteral calculi
Since the first published description of feline ureteral calculi in 1991 we’ve reached the point where it has been described as the commonest cause of azotaemia in cats. If you prospectively scan a lot of azotaemic or non-specifically poorly cats a significant proportion will have ureteral calculi.
Typical acute cases are poorly cats with abdominal pain, malaise and vomiting. Azotaemia is variable depending on the status of the contralateral kidney. Many of these patients have serial episodes of ureteral obstruction. Most of these calculi do eventually pass and, depending on how long that takes, the affected kidney is left with a variable degree of permanent impairment. During these early episodes, if one kidney has good function then azotaemia may not be apparent. Later, if one kidney is already impaired, when the second one obstructs then azotaemia develops. This often manifests as a small-kidney, big-kidney scenario. The small one being previously affected and the large one acutely so.
Acutely presenting ureteral calculi are a therapeutic dilema. Many of them may wash out with diuresis but, after a few days, the risk of permanent renal damage rises. Whilst ureterotomy is an option it is associated with a relatively high incidence of complications (urine leakage, stricture). Some of these cats have multiple calculi and/or will suffer repeated bouts of obstruction. Stenting is almost certainly better where available.
Hydronephrosis occurs where a ureteral calculus does not spontaneously pass and becomes chronic:
I believe that, in human medicine, uretero-nephrectomy would be the usual course of action. Some animals undoubtedly cope without surgery. However, it is difficult to be sure that they are not painful and there is a risk of pyelonephritis: